Lack of magnesium contributes to osteoporosis. Controlling and maintaining magnesium homeostasis is a useful intervention for maintaining bone integrity.

Controlling magnesium homeostasis appears to be crucial for bone health. If the body does not get the daily necessary amount of magnesium, osteoporosis is inevitable. In the last few years, more and more people have been confronted with this disease. Unfortunately, more and more young people are suffering from this disease, which is the result of poor nutrition. Pizza, hamburger, burek and pastries do not contain magnesium, and that is why we need to take care more and more for the youngest. The fact is that if this kind of diet continues, the list of modern diseases will move to the youngest ones.

Based on experimental and epidemiological studies, low levels of magnesium have harmful effects on bones. And that’s a fact. Magnesium deficiency contributes to osteoporosis through a direct effect on the formation of crystals in bone cells and indirectly by affecting the secretion and activity of parathyroid hormone as well as by promoting low-grade inflammation.

Overall, the control and maintenance of magnesium homeostasis is a useful intervention to maintain bone integrity.

Osteoporosis is a multifactorial disease characterized by loss of bone mass due to marked deterioration of bone microarchitecture. What does that mean? This tells you that for many years you did not feed your body with the necessary vitamins and minerals, and because of these multiple factors that did not occur in your body, you became ill.

Physiologically, bone is continuously renewed by joint and coordinated interactions between osteoclasts, cells primarily involved in bone resorption, and osteoblasts, which ensure bone formation and mineralization. Osteoporosis results from an imbalance between bone deposition and resorption. By doing so alone, you can be sure that for many years you have consumed much more calcium than magnesium and brought deposition and resorption into imbalance. Calcium and magnesium must always be in the same ratio in the body. How many dairy products do you eat a day? And how many snacks contain magnesium?

The resulting decrease in bone mass increases the risk of fractures, especially hip and spine fractures, which are associated with significant pain and suffering, disability. But since we are perfectly designed machines for movement, you can still turn back the clock and replace the reduced bone marrow by adding magnesium to your body.

Osteoporosis affects millions of people worldwide, mostly postmenopausal women. There are several reasons why this is so: pregnancy, childbirth, hormones, menstruation, menopause. We women go through so many different phases in our lives that we could say that our life cycle resembles a year with all its seasons. Spring, summer, autumn and winter

Osteoporosis therapies are available and fall into two classes, anabolic drugs that induce bone formation and anti-resorptive drugs that slow bone resorption.
But all these medicines still did not replace the main cause, which is magnesium deficiency in the body.

In particular, a significant association was found between bone density and magnesium intake, an essential micronutrient with a wide range of metabolic, structural and regulatory functions.

About 60% of total magnesium is stored in bone. One third of skeletal magnesium is found in cortical bone or on the surface of hydroxyapatite or in the hydration shell around the crystal. It serves as a reservoir of exchangeable magnesium, which is useful for maintaining the physiological extracellular concentration of cations. Bone magnesium levels are related to total magnesium stores. Therefore, superficial bone increases with magnesium loading.
Magnesium is necessary for all living cells, including osteoblasts and osteoclasts. Intracellularly, magnesium is vital for numerous physiological functions.

Several direct and indirect mechanisms contribute to the effects of low magnesium on bone density. Magnesium deficiency quickly leads to hypomagnesia, which is partially buffered by the mobilization of surface magnesium from bone. In addition, the newly formed crystals are structured differently in people with magnesium deficiency than in those who are not deficient, and this affects bone stiffness. It should also be remembered that low magnesium intake slows down cartilage and bone differentiation, as well as matrix calcification.